Dab tsi yog bilirubin hauv cov ntshav? Yog vim li cas rau qhov nce

Cov txheej txheem:

Dab tsi yog bilirubin hauv cov ntshav? Yog vim li cas rau qhov nce
Dab tsi yog bilirubin hauv cov ntshav? Yog vim li cas rau qhov nce

Video: Dab tsi yog bilirubin hauv cov ntshav? Yog vim li cas rau qhov nce

Video: Dab tsi yog bilirubin hauv cov ntshav? Yog vim li cas rau qhov nce
Video: Mus ua nyab rau yim hmoob tsis zoo 7/27/2017 2024, Kaum ib hlis
Anonim

Dab tsi yog bilirubin? Nws yog lo lus nug no uas cov neeg mob nug thaum muaj tus nqi siab ntawm cov tshuaj no hauv kev kuaj ntshav. Lo lus teb rau lo lus nug no yooj yim. Nws paub tias bilirubin yog cov kua tsib xim uas tshwm sim thaum lub sij hawm tawg ntawm cov khoom xws li hemoglobin, myoglobin thiab cytochrome.

Yuav kom paub qhov ua rau hyperbilirubinemia, koj yuav tsum sab laj nrog kws kho mob thiab ua qhov kev kuaj mob uas siv cov txheej txheem niaj hnub tshuaj xyuas lub cev, nrog rau ua raws li kev kho mob raws li kws kho mob tau sau tseg.

txheej txheem tsim ntawm bilirubin thiab nws cov nyhuv ntawm lub cev

Tom qab kuaj ntshav biochemical, tus kws kho mob tuaj yeem qhia tus neeg mob tias nws tau nce bilirubin. Nws txhais li cas? Qhov no yog kua tsib pigment. Yog tias nws qhov taw qhia siab, ces cov tshuaj tuaj yeem muaj cov tshuaj lom rau txhua lub paj hlwb. Tsis tas li ntawd, nws tseem yog qhov tseem ceeb hauv kev txhim kho encephalopathy, nuclear jaundice.

Cov txheej txheem ntawm kev sib txuas, uas yog, cov tshuaj khi ntawm cov xim (dawb hom), tshwm sim hauv daim siab cov ntaub so ntswg. Kev tawg ntawm cov qe ntshav liab, uas tshwm sim nws tus kheej hauv tus po, muaj peev xwm ua kom muaj kev tso tawm ntau ntawm cov xim los ntawm cov leeg nrog cov ntshav ntws mus rau hauv daim siab nws tus kheej. Biochemical "kev khi", kev hloov pauv hauv bilirubin hauv daim siab hlwb, piav qhia txog qhov txo qis ntawm qhov txaus ntshai ntawm txhua lub cev thiab cov ntaub so ntswg.

Ntau hom bilirubin

Txuas ntxiv lub ntsiab lus ntawm bilirubin yog dab tsi, nws yuav tsum tau muab sau tseg tias nws tshwm sim nyob rau hauv ntau hom bile feem:

  • ncaj bilirubin;
  • indirect bile pigment.

Kev kuaj ntshav yuav qhia tau tias tag nrho cov bilirubin nce siab. Qhov no txhais tau li cas tuaj yeem nkag siab tau raws li hauv qab no: thaum lub sijhawm kawm biochemical ntawm cov ntshav cov ntsiab lus, qhov ntim ntawm cov kua tsib xim yog suav tag nrho rau ob feem (tag nrho). Thiab nrog lub ntim loj lossis muaj cov cim qhia, kev kawm txog cov ntsuas ntawm txhua feem (indirect thiab ncaj qha) yuav tsum tau muab cais los ntawm ib leeg.

Indirect bilirubin ib txwm tsim: txog li 300 milligrams ntawm cov khoom raug tso tawm txhua hnub thaum lwj. Thaum dhau los ntawm cov hlab ntsha hauv hom tsis ncaj qha ntawm daim siab cov ntaub so ntswg, bilirubin pib hloov pauv vim muaj cov txheej txheem biochemical. Lawv xaus nrog conjugation nrog acid thiab cov tsos ntawm bilirubin.

Cov xim tsis ncaj nraim yog tshuaj lom rau cov ntaub so ntswg thiab lub cev. Nrog kev nkag mus tsis tiav rau hauv cov hlwb, nws pib sib txuas nrog cov rog - qhov no ua rau muaj kev cuam tshuam ntawm cov txheej txheem ntawm tes ua pa thiab cov metabolism. Protein pib tsim, poov tshuaj ions raug thauj.

High concentration (indirect form) yog suav tias yog tshwj xeeb tshaj yog txaus ntshai rau cov ntaub so ntswg hlwb. Vim tias qhov xwm txheej no, nws yog qhov yuav tsum tau xav txog cov qauv ntawm bilirubin nyob rau hauv ntau pawg hnub nyoog ntawm tib neeg.

Kev nce qib bilirubin
Kev nce qib bilirubin

Ntau yam txiaj ntsig ntawm cov kua tsib xim hauv cov menyuam mos

Nyob rau hauv cov menyuam mos, bilirubin tseem ceeb thiab yuav tsum tau tswj. Nws yog qhov tseeb tias qhov siab tshaj plaws ntawm cov xim no hauv cov ntshav, uas tsis ua rau muaj kab mob thiab kev ua haujlwm tsis zoo, tuaj yeem pom hauv cov menyuam yaus. Qhov no yog vim cov txheej txheem ntawm kev tawg sai ntawm cov qe ntshav liab hauv tus menyuam cov ntshav.

Yog li dab tsi yog bilirubin? Txuas ntxiv los teb cov lus nug no, nws yog ib qho tsim nyog yuav tsum nco ntsoov cov cim nquag hauv cov menyuam yug tshiab. Cov xim daj ntawm daim tawv nqaij thiab qhov muag nyob rau thawj lub lim tiam tom qab yug me nyuam yog qhov tshwm sim ntawm ib ntus siab bilirubin cov ntsiab lus hauv cov ntshav. Nws paub tias nws cov ntsiab lus txawv nyob ntawm hnub nyoog qeb.

Tus txheej txheem ntawm kev tso cov kua tsib ntau ntau pib thaum tus menyuam muaj hnub nyoog ib hlis. Tom qab ntawd, tus qauv ntawm bilirubin tau mus txog, thiab nws sib haum mus rau tus nqi ntawm cov neeg laus. Ntxiv mus, yog tias qhov normalization ntawm cov tshuaj no tau tshwm sim, ces qhov no tsis yog vim li cas tsis kam mus kuaj ntxiv. Nws tsim nyog txheeb xyuas qhov laj thawj.

Ua rau hyperbilirubinemia hauv cov menyuam mos

Qib tag nrho bilirubin hauv cov menyuam yug tshiab yog qhov txawv ntawm cov neeg laus. Qhov no yog vim lub fetus hemoglobin ntau dhau.

Rau cov menyuam mos tag nrho, ib txwm muaj bilirubin yog 253 mmol / l, thiab rau cov menyuam mos uas tsis yog menyuam yaus nws yog 163-167 mmol / l.

Nyob rau hauv cov me nyuam yaus muaj teeb meem tshwm sim los ntawm kev tsim cov khoom zoo li no. Muaj ntau theem ntawm cov kua tsib xim nyob rau hauv lub xub ntiag ntawm neonatal jaundice, kab mobkab mob siab ntau yam.

Nco ntsoov nco ntsoov cov laj thawj hauv qab no rau cov ntsiab lus siab ntawm bilirubin:

  • daim siab pathology;
  • Metastasis ntawm cov qog nyob hauv lwm lub cev;
  • vitamin B deficiency12;
  • Dubin-Johnson syndrome - tsis zoo tso tawm ncaj qha bilirubin rau hauv cov kua tsib;
  • ib tus mob tshwm sim los ntawm qhov tsis tuaj lossis tsis muaj cov khoom siv tshwj xeeb - cov txheej txheem ntawm kev sib txuas cov kua qaub: bilirubin thiab glucuronic acid cuam tshuam.

Cov ntsiab lus siab ntawm cov kua tsib xim tau sau tseg hauv Gelbert's syndrome thaum muaj hnub nyoog sib txawv, tab sis ntau zaus hauv cov menyuam yug tshiab.

Jaundice nyob rau hauv cov me nyuam mos
Jaundice nyob rau hauv cov me nyuam mos

Kev xeem ib txwm rau cov hnub nyoog sib txawv

Yuav kom paub meej txog tus nqi ntawm bilirubin, koj tuaj yeem xav txog cov lus hauv qab no:

Bilirubin feem

In µm

Los ntawm 0 txog peb hnub Peb mus rau rau hnub 1 hli thiab laus dua Tus neeg laus
Nruas 0, 5-10, 1(10, 2 max) 1-12, 4 0 to 5, 1(max) 0-5, 1(max)
General 24-190 28 - 209 (max 210) 3, 5- max. 20, 4 8 - max. 20, 5
Indirect 23, 4(5) - 180 (Max) 27-198 (Max) Max 16, 4-16, 5 Max 16.5

Nyob hauv qee cov kab mob hauv lub cev, cov ntsiab lus ntawm cov kua tsib xim hauv cov ntshav yuav siab dua li cov qauv ntawm bilirubin qhia hauv lub rooj.

Qhov tseem ceeb ua rau muaj cov kua tsib ntau ntxiv rau cov neeg laus

Kev kuaj ntshav tsis zoo, qhia tias ntau dhau ntawm cov qauv ntawm bilirubin ntawm ob feem - tsis muaj kev sib txuas thiab ncaj qha, yuav tsum ceeb toom rau tus neeg mob. Lub pathological pib ntawm hyperbilirubinemia yog qhov ntim ntawm cov ntshav sib npaug ntawm 34 microns ib liter ntawm cov ntshav. Ntawm qhov concentration no, jaundice pib tshwm sim.

qib siab lossis qis ntawm bilirubin hauv cov ntshav yog cuam tshuam nrog cov kab mob. Feem ntau manifested raws li nram no: ib tug loj tsub zuj zuj ntawm bile pigments pib nyob rau hauv cov proteins ntawm lub qhov muag, lub mucous nto. Qhov no yuav ua rau muaj cov xim daj sib xws.

Txawm li cas los xij, kev ntseeg siab, kab mob siab A, tshwm sim los ntawm hyperbilirubinemia, tsis muaj feem cuam tshuam rau tus kab mob siab B, txawm hais tias nws tuaj yeem tshwm sim raws li cov tsos mob ntawm tus kab mob no. Cov ntsiab lus siab ntawm cov khoom, tshwj xeeb tshaj yog thaum nws tshaj qhov kev cai, suav hais tias yog ib qho cim uas ua rau muaj kev phom sij rau tus neeg mob lub neej.

Nws yuav tsum tau muab sau tseg tias muaj ob feem ntawm bilirubin hauv cov ntshav - dawb thiab khi. Cov kab mob nrog rau qib siab ntawm cov kua tsib ncaj qha yuav tsum tau txiav txim siab:

  • mob kis kab mob siab A, B,
  • mononucleosis, zoo li kab mob siab thib ob;
  • daim ntawv tiv thaiv kab mob siab C;
  • kab mob siab ntawm kab mob hauv cov xwm txheej xws li leptospirosis;
  • teeb meem tom qab noj tshuaj lom, tshuaj: tshuaj tsis yog tshuaj steroidal tshuaj tiv thaiv kab mob, tshuaj tiv thaiv hormonal thiab lwm yam;
  • gestational jaundice hauv cov poj niam thaum cev xeeb tub;
  • qog hlav hauv daim siab.

Kev sib txawv ntawm cov qauv ntawm cov tshuaj no hauv cov ntshav tuaj yeem ua rau muaj kev txawv txav ntawm caj ces thiab tus mob uas yog qhov tshwm sim ntawm jaundice.

Kab mob siab ua rau mob bilirubinemia
Kab mob siab ua rau mob bilirubinemia

Cov cim qhia ntawm hyperbilirubinemia

Yog tias muaj kev ua txhaum ntawm daim siab ua haujlwm uas tiv thaiv kev sib txuas, kev tsim cov kua tsib xim hauv lub cev thiab cov kua tsib tawm, qhov nce hauv bilirubin tshwm sim. Ib qho ntawm cov cim qhia ntawm kev nce nqi ntawm cov khoom no, uas tau kuaj pom thaum lub sijhawm tshuaj ntsuam, suav tias yog kev ua txhaum cai hauv lub cev.

Yellow tint los ntawm cov kua tsib xim tshwm rau ntawm qhov muag dawb, thiab tom qab ntawd ntawm qhov ncauj mucosa. Tom qab ntawd, nws pib tshwm rau pem hauv ntej, ntawm qhov chaw ntawm lub xib teg thiab ntawm lub hauv siab. Qhov no tseem npog lwm qhov ntawm lub cev. Cov tsos mob tshwm sim heev uas ua rau mob jaundice thaum lub sij hawm hyperbilirubinemia yog pruritus.

Lub xub ntiag ntawm jaundice tsis tas yuav tsum tau nce siab bilirubin. Ib qho zoo sib xws ntawm daim tawv nqaij pigmentation tuaj yeem tshwm sim nws tus kheej nrog cov khoom loj ntawm cov khoom - carotene, piv txwv li, nrog ntau dhau ntawm carrots, txiv lws suav. Cov xim daj ntawm daim tawv nqaij yog cov tsos mob ntawm ntshav qab zib mellitus, hypothyroidism. Lub cim tshwj xeeb ntawm tus mob no yog cov xim ib txwm muaj ntawm sclera ntawm ob lub qhov muag.

siab tsis muaj khi thiab ncajbilirubin: cov laj thawj tseem ceeb

Cov ntsiab lus siab ntawm cov feem ntau yog qhov tshwm sim ntawm cov kab mob (cov kab mob) uas suav tias yog lub hauv paus ua rau hyperbilirubinemia. Xav txog cov ntsiab lus no kom ntxaws ntxiv:

  • dhau cov ntsiab lus ntawm cov ntshav xws li eritrocit, ua kom cov txheej txheem ntawm kev puas tsuaj;
  • tawm cov kua tsib (yam tsis pub dawb);
  • kev ua txhaum hauv kev ua thiab tshem tawm cov xim.
  • ntau cov qe ntshav thiab lawv kev puas tsuaj.

Qhov tseem ceeb tshaj plaws kom nkag siab yog tias yog qhov ncaj qha bilirubin nce siab, ces koj yuav tsum mus ntsib kws kho mob tam sim ntawd. Qhov siab ntawm kev tawg ntawm cov qe ntshav (erythrocytes) tuaj yeem ua rau muaj kev loj hlob ntawm cov kab mob jaundice xws li hemolytic. Nws yog nrog los ntawm kev tsim cov khoom loj ntawm indirect bilirubin. Cov xwm txheej zoo sib xws tshwm sim nws tus kheej hauv ntau yam pathologies, suav nrog kev hloov pauv hauv cov qe ntshav liab thiab hemorrhage (hematoma, pulmonary infarction). Kev ua txhaum cai yuav tshwm sim yog tias muaj teeb meem xws li:

  • kis kab mob;
  • malignant qog;
  • npau taws.

Yog tias muaj qhov tsis sib xws hauv cov khoom tseem ceeb xws li hom ntshav ntawm tus neeg pub dawb thiab tus neeg txais, ces jaundice tshwm. Tib lub sijhawm, cov tsos mob hauv qab no tau sau tseg:

  1. Zaj daj daj rau cov tawv nqaij dawb ntawm lub qhov muag, mucous membranes thiab tawv nqaij.
  2. tawv nqaij daj vim ntshav ntshav.
  3. Muaj qhov ntim loj ntawm qee lub cev - tus po. Muaj mob nyob rau sab laug hypochondrium.
  4. Cov xim dub ntawm cov zais zis xws li tso zis thiab quav vim yogCov ntsiab lus siab ntawm urobilin.

Kev noj qab haus huv tsis zoo yog cuam tshuam nrog cov pa oxygenation tsis zoo - mob hauv lub taub hau, lub plawv dhia ceev, qaug zog siab, nrog rau qhov nce hauv lub cev kub rau cov txiaj ntsig subfebrile.

Nruas thiab lub hlwb puas

Kev tshuaj xyuas tsis zoo yuav qhia tau tias muaj kev puas tsuaj rau lub paj hlwb thiab lub cev. Cov mob no yog hais txog kernicterus. Hauv cov neeg laus, qhov sib txawv ntawm cov qauv hauv kev tshuaj ntsuam yuav qhia tau tias muaj kab mob xws li:

  • cholecystitis;
  • kev hloov pauv ntawm bilirubin metabolism;
  • kab mob siab;
  • mononucleosis;
  • cirrhosis ntawm daim siab;
  • mob hauv siab;
  • tshuaj lom hemolysis;
  • hemolytic anemia;
  • qog nqaij hlav hauv pancreas;
  • incompatibility ntawm plasma pawg thaum lub sijhawm hloov pauv;
  • tshuaj tiv thaiv tsis zoo rau cov kab mob siv, tshuaj tiv thaiv kab mob thiab lwm yam tshuaj.

Cov ntsiab lus qis ntawm cov kua tsib xim hauv cov ntshav tau kuaj pom tsis tshua muaj. Nws yog feem ntau cuam tshuam nrog kev siv mus sij hawm ntev ntawm cov vitamin C, phenobarbital.

Kev puas tsuaj rau lub paj hlwb
Kev puas tsuaj rau lub paj hlwb

teeb meem digestinal

Kev tsis txaus ntawm cov ntsiab lus ntawm lub gallbladder tuaj yeem ua rau hyperbilirubinemia. Nyob rau hauv lub xub ntiag ntawm xws li ib tug teeb meem, cov kua tsib tivthaiv tsis tawm hauv lub cev thiab xa mus rau cov hlab ntsha. Vim li no, jaundice tsim. Feem ntau cov laj thawj uas ua rau muaj kev cuam tshuam cov dej ntws tawm muaj xws li hauv qab no:

  • qhov pom ntawm qhov nqaim ntawm cov ducts tshem tawm cov kua tsib, thiabKuj tseem muaj cov pob zeb nyob hauv lawv, tsim, pathologies hauv cov ntaub so ntswg, kab mob cab;
  • mob;
  • kab mob nyob rau hauv lub biliary ib ntsuj av ntawm ib tug inflammatory xwm, uas provokes sclerosis ntawm lub ducts.

tshwm sim hauv cov ntshav tsis zoo thiab cov kab mob hauv lub cev thaum yug los.

YGilbert's Syndrome

Ib txwm nce bilirubin hauv cov ntshav? Yog li ntawd nws yog kab mob siab congenital? Qhov kev xaiv no kuj tau txais txiaj ntsig zoo, thiab qhov tshwm sim tseem ceeb ntawm tus kab mob no yog benign hyperbilirubinemia. Muaj cov tsos mob ntawm jaundice, txuam nrog lipofuscins hauv hepatocytes.

Gilbert's syndrome tau piav qhia thaum ntxov li 1900 - nws yog hu ua yooj yim tsev neeg cholemia, uas yog, benign tsev neeg uas tsis yog hemolytic hyperbilirubinemia.

Tus kab mob no tsawg. Vim li no, benign hyperbilirubinemia yuav tsum tau suav nrog hauv kev tshuaj ntsuam xyuas rau cov neeg mob uas muaj ntshav siab xim xim.

Daj dawb ntawm qhov muag
Daj dawb ntawm qhov muag

daim duab kho mob: cov tsos mob

Nyob rau hauv Gilbert's syndrome, bilirubin pib nce siab vim muaj kev cuam tshuam xws li:

  • capture;
  • thauj;
  • kev sib txuas hauv hlwb.

Tus mob no tshwm sim vim muaj qhov tsis zoo enzyme. Vim tias qhov ua haujlwm tsis zoo ntawm cov enzyme no, lub siab tuaj yeem ua rau poob tag nrho xws li kev ua haujlwm tsis zoo. Muab lub pathogenesis ntawm tus kab mob, nws yuav tsum tau muab sau tseg tias nws yog txuam nrog impaired thauj muaj nuj nqi ntawm cov proteins uas xa bilirubin (tag nrho) rau microsomes.

Ntau zausTus kab mob pib tshwm sim nyob rau hauv cov menyuam yaus thaum muaj hnub nyoog 14 xyoo los ntawm lub caij nyoog thiab nrog rau ib tug neeg tag nrho nws lub neej. Kwv yees li 30% ntawm cov neeg tsis yws txog lawv txoj kev noj qab haus huv. Nws yuav siv sij hawm ntev heev nyob rau hauv lub xeev ntawm kev zam txim, thiab nyob rau tib lub sij hawm tsis muaj kev hloov nyob rau hauv ntxoov ntxoo ntawm daim tawv nqaij thiab ob lub qhov muag uas cov txheeb ze pom. Feem ntau, xws li tus mob no yog ib qho xwm txheej uas tshwm sim thaum kuaj tus neeg mob.

Feem ntau, tus kab mob muaj tag nrho cov tsos mob:

  1. hom kab mob jaundice - cuam tshuam nrog ntau tshaj ntawm unconjugated bilirubin.
  2. Cov tsos mob Asthenoneurotic.
  3. Lub xub ntiag ntawm qhov mob thiab cuam tshuam hauv kev ua haujlwm ntawm lub plab. Muaj me ntsis hysteria ntawm sclera.

Nyob rau hauv txoj kev loj hlob ntawm Gilbert's syndrome, bilirubin hauv cov neeg laus siab dua 21 mmol / l.

Hyperbilirubinemia hauv cov poj niam cev xeeb tub

Qhov nce ntawm qhov ntsuas no hauv pawg neeg no muaj ntau yam laj thawj, tab sis muaj ntau tshaj plaws ntawm lawv. Piv txwv li, thaum cev xeeb tub, tej zaum yuav muaj me ntsis nce ntawm cov ntsiab lus ntawm cov khoom txuam nrog qhov loj ntawm tus me nyuam hauv plab. Vim tias qhov kev hloov pauv me me thiab lub cev muaj zog, lawv tsis pom hauv kev tshuaj ntsuam ntshav.

Nrog cov bilirubin ntau dhau hauv cov ntshav hauv cov poj niam, qhov muaj peev xwm ntawm cov kab mob txaus ntshai lossis kev ua haujlwm tsis zoo ntawm cov kua tsib lub cev yog qhov tseem ceeb hauv qhov xwm txheej no.

Muaj ntau yam kab mob thiab cov xwm txheej uas ua rau muaj kev hloov pauv hauv kev ua haujlwm ntawm cov xim no. Lawv yog cov txaus ntshai, yog vim li cas nws tseem ceeb heev kom mus ntsib kws kho mob raws sij hawm.

Qhov nce ntawm qhov ntim ntawm bilirubin hauv cov ntshav tsis tshwm simTsuas yog thaum muaj mob, tab sis kuj yog vim li cas lub cev muaj feem xyuam nrog kev xeeb tub:

  • stagnation ntawm cov kua tsib - lawv qhov ua rau yog kev loj hlob ntawm tus menyuam hauv plab thiab lub cev, uas ua rau tus poj niam sab hauv;
  • qhov muaj tus mob zoo li no thaum ntxov toxicosis tau sau tseg hauv ntau tus poj niam hauv thawj peb lub hlis twg ntawm cev xeeb tub;
  • cev xeeb tub cholestasis (intrahepatic) - tshwm sim nws tus kheej tom qab, thiab pib kis thaum yug menyuam yam tsis tas yuav muaj kev kho mob.

Dystrophy ntawm daim siab ntawm cov poj niam cev xeeb tub kuj yog ib qho pathology tsis tshua muaj tshwm sim uas tshwm sim thawj zaug hauv cov poj niam cev xeeb tub uas yug menyuam thawj zaug.

Anemia hemolytic - ua rau yug ntxov lossis tuag ntawm tus menyuam hauv plab. Qhov thib ob ua rau tus kab mob yog kab mob siab. Kev loj hlob tsis zoo ntawm cov placenta kuj tseem ua tau, uas ua rau ncua kev loj hlob ntawm fetus. Nws kuj tseem muaj peev xwm ua rau tus menyuam hauv plab kis tau tus mob xws li kab mob siab E hauv plab.

Ib tus kab mob xws li cholecystitis tuaj yeem ua rau kev loj hlob ntawm preeclampsia nyob rau theem tom ntej. Nws yog qhov txaus ntshai rau leej niam thiab tus menyuam mos uas muaj cov tsos mob hauv qab no: edema, ntshav siab, ua rau intrauterine loj hlob retardation.

High bilirubin ua rau muaj kev phom sij rau kev yug menyuam thiab qhov tshwm sim, tab sis ua ntej panicking, nws yog ib qho tsim nyog yuav tsum rov kuaj xyuas biochemical, vim lwm qhov laj thawj yuav yog kev npaj tsis raug ntawm tus poj niam rau kev tshuaj xyuas. Nws yog ib qho tsim nyog yuav tsum ua tib zoo mus txog cov teeb meem ntsig txog kev xa cov ntawv xeem.

Muaj cov xwm txheej thaum lub sijhawm tshuaj ntsuam, nws tau qhia tiastag nrho bilirubin hauv cov ntshav yog tsawg heev. Qee zaum qhov no tsis yog vim lub raum tsis ua haujlwm ntev, tab sis kuj rau lwm yam laj thawj.

Cov kab mob ntawm lub plab zom mov
Cov kab mob ntawm lub plab zom mov

Dab tsi ua rau cov kua tsib qis qis?

ua rau muaj kev phom sij tuaj yeem yog kab mob xws li:

  • mob leukemia;
  • tuberculosis;
  • aplastic anemia;
  • kev npaj phem.

Leukemia yog qhov ua rau qis bilirubin hauv cov ntshav. Nws tseem muaj peev xwm hais tias kev haus luam yeeb los ntawm tuberculosis ua rau kev soj ntsuam tsis zoo. Cov kab mob no suav tias yog qhov txaus ntshai, thiab kev kuaj xyuas thiab kho raws sijhawm tuaj yeem zam qhov teeb meem txaus ntshai.

Aplastic anemia

Kev ua haujlwm tsis zoo yog vim daim ntawv no ntawm ntshav ntshav, uas yog ib yam kab mob tsawg thiab nyuaj uas yuav tsum tau kho thiab kho raws sijhawm.

Yog li dab tsi yog bilirubin? Rov qab mus rau qhov teeb meem no, nyob rau hauv cov ntsiab lus ntawm aplastic anemia, nws yuav tsum tau muab sau tseg tias nrog rau cov kab mob no muaj ib tug txo nyob rau hauv cov ntsiab lus ntawm cov ntshav liab thiab, raws li, muaj ib tug nyiam rau lawv kev puas tsuaj thiab txo cov ntsiab lus ntawm cov ntshav liab. bilirubin.

mob plawv ischemic

Nws kuj ua rau muaj kev cuam tshuam ntawm plab hnyuv thiab, vim li ntawd, kev sim tsis zoo.

Hais txog cov neeg mob uas muaj hnub nyoog tshaj 60 xyoo, nws yuav tsum tau muab sau tseg tias kev txo qis ntawm cov kua tsib xim yog yam ntxwv ntawm IHD (mob plawv) ntawm ntau hom.kev siv zog. Clinically, tus kab mob no tshwm sim nrog cov tsos mob ntawm tsawg bilirubin.

Nws tsim nyog sau cia tias kev npaj tsis raug rau kev pub ntshav rau kev tshawb fawb kuj tuaj yeem ua rau.

Khau los ntawm cov xim
Khau los ntawm cov xim

Lub Tswv Yim rau Kev Xeem Ua tiav

Nws raug nquahu kom koj paub koj tus kheej nrog qee cov lus qhia tshwj xeeb uas yuav tsum tau ua raws tam sim ua ntej noj ntshav rau kev tshuaj ntsuam biochemical:

  1. Tsis txhob noj 12 teev ua ntej kuaj.
  2. txwv tsis pub ua lub cev hnyav. Qhov no yuav cuam tshuam qhov tshwm sim ntawm kev tshuaj ntsuam.
  3. Tsis txhob haus dej haus uas muaj caffeine lossis cawv.
  4. Nws tau thov kom txwv cov tshuaj uas cuam tshuam rau bilirubin.

Ua ntej ua qhov ntsuas, nws raug nquahu kom tsis txhob ua lub cev hnyav, vim tias cov txiaj ntsig yuav tsis raug nrog lawv.

Cov lus pom zoo rau kev cuam tshuam nrog siab bilirubin

Koj yuav tsum ua raws li tag nrho cov lus qhia los ntawm tus kws kho mob nrog kev tshuaj xyuas tsis zoo, vim tias yog tias koj tsis siv cov nyiaj tau los thiab tsis tiv tauj cov kws kho mob tshwj xeeb txog qhov teeb meem no, koj tuaj yeem ua rau muaj kev cuam tshuam loj.

Lub hom phiaj tseem ceeb ntawm kev kho cov kab mob thiab lwm yam ua rau hyperbilirubinemia yog kom ua tiav qib ib txwm lossis qib siab ntawm cov tshuaj no. Nws yog txiav txim los ntawm tus kws kho mob thiab nws tsis tas yuav tsum nyob rau hauv 21 mmol / l - siab dua qhov tseem ceeb nyob rau hauv lub xub ntiag ntawm tej yam kab mob. Txawm li cas los xij, tus mob yog qhov txaus ntshai thaum bilirubin hauv cov ntshav nce siab. Yog vim li cas rau qhov kev ua txhaum cai noyuav tsum tau kuaj xyuas thiab kho raws sijhawm.

Pom zoo: