Cov ntaub ntawv keeb kwm: hom 2 mob ntshav qab zib mellitus, theem decompensation, subcompensation, chav kawm hnyav, insulin-dependant, insulin-yuav tsum tau, ntshav qab zib polyne

Cov txheej txheem:

Cov ntaub ntawv keeb kwm: hom 2 mob ntshav qab zib mellitus, theem decompensation, subcompensation, chav kawm hnyav, insulin-dependant, insulin-yuav tsum tau, ntshav qab zib polyne
Cov ntaub ntawv keeb kwm: hom 2 mob ntshav qab zib mellitus, theem decompensation, subcompensation, chav kawm hnyav, insulin-dependant, insulin-yuav tsum tau, ntshav qab zib polyne

Video: Cov ntaub ntawv keeb kwm: hom 2 mob ntshav qab zib mellitus, theem decompensation, subcompensation, chav kawm hnyav, insulin-dependant, insulin-yuav tsum tau, ntshav qab zib polyne

Video: Cov ntaub ntawv keeb kwm: hom 2 mob ntshav qab zib mellitus, theem decompensation, subcompensation, chav kawm hnyav, insulin-dependant, insulin-yuav tsum tau, ntshav qab zib polyne
Video: Qhia tsim txoj Kev MUAJ HMOOV zoo rau koj ( Lus tsim nuj, leej twg tsis tau mloog yuav tu siab). 2024, Lub Xya hli ntuj
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Tej zaum tsis muaj coob tus neeg txaus siab rau lawv cov keeb kwm kho mob. Hom 2 mob ntshav qab zib mellitus tau teev tseg hauv nws tuaj yeem ua rau muaj kev poob siab ntau dua. Tab sis nws puas tsim nyog tso tseg thiab poob siab? Tus kab mob no txaus ntshai npaum li cas, thiab yuav ua li cas nrog nws - peb yuav txheeb xyuas nws.

Mob ntshav qab zib mellitus - Dab tsi yog qhov txaus ntshai?

Kev kho mob keeb kwm - Hom 2 mob ntshav qab zib mellitus
Kev kho mob keeb kwm - Hom 2 mob ntshav qab zib mellitus

Tib neeg feem ntau hnov txog ntshav qab zib, tab sis tsawg tus paub tias nws yog dab tsi. Nws yuav tsum raug sau tseg tias lub tswv yim ntawm "mob ntshav qab zib mellitus" yog ib qho kev sib sau ua ke. Nyob rau hauv lub sij hawm no, ntau yam kab mob ntawm lub endocrine system yuav ua ke. Qhov ua rau ntawm lawv qhov tshwm sim muaj ntau heev, tab sis, raws li txoj cai, tus kab mob yog raws li endocrine ntshawv siab, thiab tshwj xeeb tshaj yog insulin deficiency. Lwm qhov tseem ceeb yog qhov tsis muaj peev xwm nqus cov piam thaj los ntawm lub siab thiab cov ntaub so ntswg hauv lub cev. Nyob rau tib lub sijhawm, nws qib hauv lub cev nce, uas, dhau los, yog qhov ua rau muaj ntau yam kabmob metabolic.

Qab Zibdiabetes - nws hom

Cov tshuaj niaj hnub paub qhov txawv ntawm ob hom kab mob no. Gradation tshwm sim nyob ntawm seb hom tshuaj insulin tsis txaus - txheeb ze lossis qhov tseeb - piav qhia txog keeb kwm ntawm tus kab mob. Hom 2 mob ntshav qab zib mellitus, raws li txoj cai, tsis cuam tshuam nrog qhov tsis txaus ntawm cov tshuaj insulin, uas yog tsim los ntawm cov txiav. Feem ntau qhov ua rau nws txoj kev loj hlob yog ib qho kev txo qis ntawm cov kabmob thiab cov ntaub so ntswg rau cov tshuaj hormones. Ntshav qab zib insulin-dependent - thawj hom - cuam tshuam ncaj qha rau insulin deficiency hauv lub cev. Qhov tsis muaj cov tshuaj hormones no ua rau lub fact tias cov piam thaj uas nkag mus rau hauv lub cev tsis ua tiav, lossis nws tshwm sim hauv qhov tsis txaus, thaum nws qib hauv cov ntshav nce ntxiv, thiab txhawm rau cawm txoj sia, cov neeg mob no raug yuam kom tau txais. Ntxiv koob tshuaj insulin.

Case keeb kwm - ntshav qab zib hom 2

Cov ntshav qab zib no tseem hu ua non-insulin dependent. Raws li kev txheeb cais, muaj ntau tus neeg mob raug mob los ntawm hom kab mob no ntau dua li thawj zaug. Nrog rau hnub nyoog, qhov kev pheej hmoo ntawm tus kab mob no yuav tshwm sim nws tus kheej nce ntau. Feem ntau, nws txoj kev loj hlob tshwm sim imperceptibly, thiab tus neeg mob tsis paub txog cov kab mob no rau lub sij hawm ntev, cov ntaub ntawv uas tso cai rau nws xav tias yuav tsis cuam tshuam txog keeb kwm ntawm tus kab mob. Hom 2 mob ntshav qab zib yog tom qab ntawd nrhiav pom raws li "kev nrhiav pom" thaum lub sijhawm kuaj niaj hnub. Qhov taw qhia tseem ceeb uas tso cai rau koj xav tias muaj cov kab mob pathology no yog kev kuaj ntshav uas cov piam thaj nce siab. Nyob rau hauv lem, thaum ib tug kho mob keeb kwm yog kos,Hom 2 mob ntshav qab zib mellitus tau muab faib ua:

  • tshwm sim hauv cov neeg mob hnyav ib txwm;
  • txheeb xyuas cov neeg mob hnyav dhau.
Cov ntaub ntawv keeb kwm - hom 2 mob ntshav qab zib mellitus - xav tau insulin
Cov ntaub ntawv keeb kwm - hom 2 mob ntshav qab zib mellitus - xav tau insulin

Txawm tias nyob rau hauv cov txheej txheem ntawm kev cob qhia, cov kws kho mob hluas ua tiav kev tshawb fawb keeb kwm ntawm tus kab mob, hom 2 mob ntshav qab zib mellitus uas tuaj yeem tshwm sim los ntawm ntau yam. Cov feem ntau yog:

  • Kev noj zaub mov zoo carbohydrates (qab zib, chocolate, muffins, qos yaj ywm) ntau thiab tsis muaj cov nroj tsuag fibers hauv kev noj haus;
  • heredity - nyob rau hauv lub xub ntiag ntawm ib tug zoo xws li pathology nyob rau hauv cov niam txiv, qhov kev pheej hmoo ntawm kev mob ntshav qab zib ntau tshaj 40%;
  • rog, tshwj xeeb tshaj yog hom visceral (cov rog rog nyob hauv lub plab feem ntau);
  • mob ntshav siab;
  • hypodynamia.

Yuav ua li cas? Pathogenesis

Cov ntaub ntawv keeb kwm piav qhia txog hom 2 mob ntshav qab zib hom 2 yog cov kab mob metabolic. Nws yog raws li insulin tsis kam - qhov tsis muaj peev xwm ntawm cov ntaub so ntswg rau ib txwm pom cov tshuaj insulin, ntawm ib sab, thiab kev ua txhaum ntawm kev tsim cov tshuaj hormones no hauv cov hlwb pancreatic, ntawm lwm qhov. Feem ntau, nrog kev nce qib ntawm cov piam thaj uas tshwm sim tam sim tom qab noj mov, lub txiav mob siab pib tsim cov tshuaj insulin. Hauv cov neeg mob ntshav qab zib hom 2, qhov no tsis tshwm sim, thiab insulin pib tsim tsuas yog thaum qib ntawm cov piam thaj ntau dhau lawm. Txawm li cas los xijmuaj cov tshuaj insulin, qhov txo qis hauv cov piam thaj hauv qhov no tsis tshwm sim rau qib xav tau. Kev nce siab tas li ntawm cov tshuaj insulin ua rau cov hlwb poob lawv lub peev xwm los teb rau nws, thiab tam sim no, txawm tias cov tshuaj hormones tswj txuas mus rau lub cell receptor, tsis muaj cov tshuaj tiv thaiv. Tawm tsam keeb kwm yav dhau los ntawm cov kev hloov pauv no, lub siab hlwb pib ua kom cov piam thaj ntau ntxiv, ntxiv rau nws cov concentration hauv cov ntshav. Tag nrho cov no tsis dhau yam tsis muaj qhov tshwm sim. Nyob rau tib lub sijhawm, nyob rau hauv kev cuam tshuam ntawm cov piam thaj ntau ntau, cov piam thaj toxicity tshwm sim, uas cuam tshuam tsis zoo rau cov hlwb ntawm cov txiav uas tsim cov tshuaj insulin, thiab, vim li ntawd, nws qhov tsis txaus yuav tshwm sim. Yog li, keeb kwm ntawm tus kab mob yog aggravated. Hom 2 mob ntshav qab zib mellitus insulin-yuav tsum tau hloov daim ntawv thaum tsis tau txais kev tswj hwm tshuaj insulin. Tus neeg mob lub cev tsis zoo.

Symptoms

Hauv chav kho mob ntshav qab zib classical, nws yog ib txwm ua kom paub qhov txawv ntawm ob pawg ntawm cov cim ntawm tus kab mob - loj thiab me. Cov tseem ceeb muaj xws li:

  • polyuria - tso zis ntau ntau (qabzib, nkag mus rau hauv nws, nce osmotic siab);
  • nqhis dej (polydyspepsia) - tus cwj pwm los ntawm qhov tsis tu ncua, xav tau haus tsis txaus;
  • tshaib plab (polydysphagia) - vim qhov tsis muaj peev xwm ntawm cov hlwb kom nqus cov piam thaj nrog kev pab ntawm insulin, lub cev xav tau lub zog tas li;
  • poob ceeb thawj - nws tshwm sim tawm tsam keeb kwm ntawm kev noj qab haus huv.

Tag nrho cov tsos mob no yog tus yam ntxwv ntawm thawj hom ntshav qab zib. Hom thib ob feem ntau yog asymptomatic lossis ua rau cov tsos mob thib ob:

  • khaus ntawm daim tawv nqaij thiab cov mucous membranes - feem ntau thaum kuaj tus neeg mob thiab xav tias muaj hom 2 mob ntshav qab zib mellitus (cov ntaub ntawv keeb kwm), tus poj niam yuav pom tias khaus khaus hauv qhov chaw mos;
  • kev qaug zog thiab mob taub hau;
  • daim tawv nqaij mob uas nyuaj kho;
  • pom kev txo qis.

Raws li txoj cai, feem ntau tsuas yog tsis xyuam xim rau cov tsos mob zoo li no, thiab tus kab mob tsuas tuaj yeem kuaj pom thaum muaj teeb meem.

Cov menyuam yaus thiab ntshav qab zib

Pediatrics. Mob ntshav qab zib mellitus hom 2, keeb kwm kho mob
Pediatrics. Mob ntshav qab zib mellitus hom 2, keeb kwm kho mob

Hmoov tsis zoo, ntshav qab zib tam sim no tau lees paub tias yog tus mob thib ob uas muaj mob ntev tshaj plaws. Qhov no yog qhov official pediatrics xav. Mob ntshav qab zib mellitus (hom 2) feem ntau yog cov kab mob txaus ntshai tshaj plaws hauv tus menyuam keeb kwm kho mob. Qhov no yog vim lub fact tias nws cov chav kawm txawv los ntawm tus kab mob ntawm ib tug neeg laus nyob rau hauv ib tug ntau ceev txoj kev loj hlob thiab feem ntau mus malignantly. Txawm li cas los xij, nyob rau hauv cov ntaub ntawv no, nws tag nrho yog nyob ntawm lub peev xwm ntawm cov niam txiv los kho tus menyuam txoj kev noj haus thiab saib xyuas cov tshuaj raws sijhawm. Tsis tas li ntawd, nws tseem ceeb heev kom kuaj tau tus kab mob hauv lub sijhawm. Yog tias tus menyuam muaj cov tsos mob hauv qab no, kev sab laj nrog tus kws kho mob endocrinologist yog tsim nyog:

  • me nyuam nqhis dej tas li;
  • mus rau chav dej ntau zaus (hauv tsev kawm ntawv lossis qib kindergarten, nws yuav tsum tau siv sijhawm los kawm ob peb zaug);
  • poob phaus;
  • Kev qaug zog ntev yog pom.

Diagnosis

Kev kuaj ntshav qab zib raws li kev kuaj zis thiab ntshav. Hauv ib tus neeg noj qab haus huv,Cov piam thaj hauv cov zis tsis raug txiav txim siab, thiab hauv cov ntshav nws qib tsis tshaj 5, 5. Rau cov txiaj ntsig zoo, cov ntshav tau muab los ntawm tus ntiv tes lossis cov hlab ntsha (zaum kawg yuav tsum tau noj tsawg kawg 8 teev ua ntej txheej txheem), tshuaj yej qab zib., kas fes lossis kua txiv yuav tsum tsis txhob haus cawv ua ntej pub dawb. Nws yuav tsum raug sau tseg tias ntshav qab zib tau kuaj pom nrog cov ntsuas saum toj no 7. Yog tias cov ntsuas ntsuas thaj tsam li ntawm 4 txog 7, ces qhov kev ua txhaum ntawm cov piam thaj hauv ntshav tau raug kaw.

Lwm qhov kev sim uas tuaj yeem kuaj pom tus kab mob no thiab qhia meej qhov tsis txaus ntseeg yog qhov ntsuas ntshav qabzib siab (GTT). Nws tso cai rau koj kom tau txais cov ntaub ntawv ntawm qib qabzib ntawm lub plab khoob, nrog rau ob teev tom qab nws noj cov piam thaj. Feem ntau, tom qab cov piam thaj, nws cov concentration yuav tsum tsis txhob siab tshaj 7.8. Qhov kev kuaj no tsis ua thaum cov piam thaj ua ntej qhov ntsuas siab tshaj 6.1, yog tias tus neeg mob muaj keeb kwm mob hnyav, phais, myocardial infarction, raug mob, thiab thaum noj. tshuaj uas ua kom cov piam thaj ntau ntxiv. Tag nrho cov ntaub ntawv no qhia txog keeb kwm kho mob.

Type 2 diabetes, subcompensation

Zoo li txhua yam kab mob, ntshav qab zib zoo li tsim thiab ua rau cov tsos mob hnyav dua. Muaj peb theem nyob rau hauv nws cov chav kawm. Lub sijhawm them nyiaj raug teeb tsa thaum tus neeg mob qhov ntsuas tau ua raws li tus qauv. Yog tias ntsuas tsis tau raws sijhawm, lossis lawv tsis coj qhov kev xav tau, ces tus kabmob kis mus rau theem ntawm subcompensation. Nyob rau tib lub sijhawm, tus neeg mob tus mob, ntawm chav kawm, tsis yog qhov zoo tshaj plaws, tab sis kuj tsis tseem ceeb. Nyob rau hnub tus neeg mob poob tsis pub ntau tshaj tsib caug grams qabzib nyob rau hauv cov zis, thiab nws cov ntshav yog hais txog 13.8,acetone tsis nyob hauv cov zis. Txoj kev loj hlob ntawm hyperglycemic coma nyob rau theem no kuj tsis yooj yim sua. Cov theem ntawm subcompensation muaj xws li ib tug nruab nrab lub xeev ntawm cov nyiaj them thiab decompensation, thaum lub sij hawm rau cov theem ntawm cov nyiaj them, lub sab sauv nqi noj cov piam thaj ntawm 7.5, cov feem pua ntawm glycated hemoglobin yog 6.5%, thiab ceev cov piam thaj tsawg dua 6.1.. Cov theem ntawm decompensation yog teem yog tias cov ntsuas tau tshaj qhov txwv hauv qab no:

  • glucose qib siab dua 9;
  • glycerated hemoglobin - ntau dua 9%;
  • fasting qabzib - ntau dua 7.

Hloov pauv ntawm cov ntsuas no thiab qhov ua tau zoo ntawm kev kho mob tau pab txhawm rau txheeb xyuas keeb kwm ntawm tus kab mob. Hom 2 mob ntshav qab zib mellitus, qhov kev mob hnyav uas nyuaj rau kev kho lossis ua rau lwm yam tshwm sim, dhau mus rau theem ntawm decompensation.

yam ua rau decompensation

Hom 2 mob ntshav qab zib mellitus, keeb kwm kho mob - prognosis
Hom 2 mob ntshav qab zib mellitus, keeb kwm kho mob - prognosis

Tau kawg, txhua yam kab mob yog tus kheej, thiab cov yam ntxwv ua rau tus kab mob hnyav dua tuaj yeem sib txawv, tab sis cov no feem ntau:

  • overeating, noj txhaum cai;
  • yuam kev hauv kev xaiv tshuaj, nws koob lossis tsis kam kho;
  • tshuaj noj rau tus kheej lossis siv cov tshuaj noj es tsis txhob siv tshuaj;
  • tsis kam hloov mus rau kev kho tshuaj insulin;
  • kab mob uas ua rau lub cev qhuav dej;
  • cov xwm txheej ntxhov siab.

Txhua yam no, tus kheej lossis ua ke, cuam tshuam rau lub xeev ntawm cov txheej txheem metabolic, thiab, vim li ntawd, muaj qhov nce ntxiv.ntsuas cov piam thaj hauv cov ntshav, uas ntes cov keeb kwm ntawm tus kab mob. Hom 2 mob ntshav qab zib mellitus, theem ntawm decompensation yog aggravated los ntawm cov tsos ntawm ntau yam teeb meem los ntawm lub cev thiab lub cev, ua hem thiab, yog hais tias tsis kho kom raug, yuav ua rau muaj kev hem thawj rau lub neej ntawm tus neeg mob. Nyob rau tib lub sijhawm, cov teeb meem tuaj yeem tshwm sim ob qho tib si hauv lub sijhawm thiab qhov tseeb hauv ib teev.

Decompensation stage

Raws li qhov concentration ntawm qabzib nce ntxiv, thiab nws txoj kev kho yuav tsum tau ntsuas hnyav dua, keeb kwm kev kho mob hnyav dua. Mob ntshav qab zib mellitus (hom 2), lub decompensation uas yog tus cwj pwm los ntawm kev pheej hmoo siab ntawm cov teeb meem, yuav tsum tau ceev faj heev. Qhov no yog vim qhov tseeb tias nws tuaj yeem nyuaj heev los kho cov qib qabzib ob leeg nrog kev pab ntawm kev noj zaub mov thiab kev siv tshuaj. Yog li ntawd, nws yog ib qho tsim nyog yuav tsum ua tib zoo saib xyuas tus neeg mob tus mob tas li, vim tias thaum tsis muaj kev kho mob thaum siv tshuaj, cov kev hloov pauv pathological hauv ntau lub cev tau tshwm sim sai heev. Yog li ntawd, cov neeg mob no xav tau kev saib xyuas kev kho mob, thiab kev saib xyuas xwm txheej ceev heev.

Kev muaj peev xwm tswj hwm koj tus kheej yog qhov tseem ceeb ntawm no. Hmoov zoo, cov tshuaj niaj hnub muaj kev xaiv txaus rau qhov no. Ntau yam kev sim thiab cov khoom siv uas txiav txim siab qib qabzib tso cai rau tus neeg mob ntshav qab zib kom kuaj tus kheej ntawm lawv tus kheej thiab, nyob ntawm qhov tshwm sim, ua qhov kho.

Cov ntaub ntawv keeb kwm, ntshav qab zib mellitus 2hom decompensation theem
Cov ntaub ntawv keeb kwm, ntshav qab zib mellitus 2hom decompensation theem

Insulin hauv kev kho mob ntshav qab zib

Raws li tus kab mob no zuj zus lawm, depletion ntawm pancreatic cells uas tsim cov tshuaj insulin kuj tsim. Cov txheej txheem no yuav tsum tau teem caij tam sim ntawm kev kho tshuaj hormone. Raws li kev tshawb fawb niaj hnub no qhia tau hais tias, qee zaum qhov no tsuas yog txoj hauv kev los tswj cov piam thaj hauv qib siab. Txawm li cas los xij, koj yuav tsum tsis txhob xav tias qhov kev teem sijhawm ntawm insulin yog rau lub neej. Feem ntau, hom 2 mob ntshav qab zib mellitus, insulin-dependant, uas nws keeb kwm kho mob, nws yuav zoo li, yuav tsum tau sau cov tshuaj insulin tas mus li, nrog rau qee qhov kev siv zog ntawm tus neeg mob, tuaj yeem txav mus rau hauv qeb thaum tswj hwm cov tshuaj no tsis tu ncua. xav tau. Hauv qhov no, ntshav qab zib mellitus hais txog cov pathologies uas tswj thiab siv zog ntawm tus neeg mob tseem ceeb heev kom ua tiav cov txiaj ntsig kho. Yog li, cov qib nyiaj them rau tus kab mob yuav nyob ntawm nws txoj kev qhuab qhia hauv kev cuam tshuam nrog kev noj zaub mov, kev saib xyuas tus kheej ntawm cov suab thaj thiab kev noj tshuaj raws sijhawm. Txawm li cas los xij, qee zaum, kev teem sijhawm ntawm insulin yog qhov yuav tsum tau ntsuas. Qhov no tshwm sim:

  • thaum cev xeeb tub;
  • mob stroke lossis plawv nres;
  • rau kev cuam tshuam kev phais;
  • rau cov kab mob sib kis ntawm tus kab mob purulent-septic;
  • nrog rau kev kuaj mob siab.

Lwm qhov laj thawj tseem ceeb rau kev teem sijhawm yuav tsum tau txhaj tshuaj insulin tuaj yeem yog qhov tsis muaj cov tshuaj hormone no, uas lees paub keeb kwm ntawm tus kabmob. ntshav qab zib mellitus 2hom, theem ntawm decompensation uas yog tus cwj pwm los ntawm qhov hnyav hnyav zuj zus thiab nce ketoacidosis, yog qhov piv txwv tseem ceeb ntawm qhov no.

Nws yuav tsum tau muab sau tseg tias niaj hnub no cov tshuaj tau nthuav dav dav ntawm kev siv cov tshuaj insulin, thiab nws tau lees paub tias qhov ua tau zoo ntawm qhov ncauj monotherapy siv sijhawm tsawg dua 6 xyoo. Tom qab ntawd koj yuav tsum tau sau ntau yam tshuaj. Raws li cov xwm txheej tshiab tshaj plaws hauv kev kho mob ntshav qab zib mellitus, kev siv cov tshuaj insulin hauv cov xwm txheej zoo li no suav tias ua tau zoo dua.

Kev nyuaj siab

Kev kho mob keeb kwm, hom 2 mob ntshav qab zib mellitus, mob ntshav qab zib polyneuropathy
Kev kho mob keeb kwm, hom 2 mob ntshav qab zib mellitus, mob ntshav qab zib polyneuropathy

Cov teeb meem hnyav ntawm theem decompensation yog cov xwm txheej uas muaj kev hem thawj rau tus neeg mob lub neej. Lawv feem ntau tshwm sim los ntawm kev nce qib ntawm cov piam thaj ntau ntxiv thiab cuam tshuam nrog kev cuam tshuam ntawm metabolic. Thaum muaj qhov xwm txheej zoo li no, nws yog ib qho tseem ceeb uas yuav tsum tau muab kev pab raws sij hawm thiab, vim tsis yog cov kws kho mob nkaus xwb, tab sis kuj cov neeg ua haujlwm saib xyuas neeg mob yuav tsum paub cov tsos mob ntawm cov mob no, cov keeb kwm kev laus ntawm hom 2 mob ntshav qab zib mellitus thiab nws cov teeb meem tau piav qhia hauv qab no.

  1. Hyperglycemic coma (hyperosmolar thiab ketoacidotic) - lawv lub ntsiab lus yog qhov kev loj hlob zuj zus. Cov laj thawj uas ua rau lawv yog cov koob tshuaj tsis txaus lossis tsis tau txais cov tshuaj insulin, kev noj zaub mov tsis zoo (kev noj zaub mov ntau dhau), kev ua haujlwm ntawm lub cev tom qab ntawm hyperglycemia, tsis muaj kev tswj cov piam thaj, cawv lossis qee yam tshuaj. Clinically lawvtshwm sim los ntawm kev nqhis dej ntau ntxiv thiab polyuria (nce cov zis) ntau hnub, lub cev qhuav dej hnyav. Arterial siab txo, tachycardia. Yog hais tias tus mob hnyav zuj zus, convulsions lossis kev nyuaj siab ntawm kev nco qab yog ua tau. Cov tawv nqaij qhuav, khawb yog ua tau, tsis hnov tsw ntawm acetone thaum exhaling. Hauv qhov no, tus neeg raug tsim txom xav tau mus pw hauv tsev kho mob sai thiab kho mob.
  2. Hypoglycemic coma - tshwm sim los ntawm kev txo qis hauv cov ntshav qabzib (tsawg dua 3 mmol / l), vim tias lub hlwb tau ntsib qhov tsis muaj cov as-ham. Tus mob no tuaj yeem tshwm sim los ntawm kev noj ntau dhau ntawm cov tshuaj insulin, hla ib qho ntawm cov pluas noj lossis kev noj zaub mov tsis raug, noj cawv lossis tshuaj. Clinically, qhov kev tawm tsam sai sai. Tus cwj pwm los ntawm hws txias ntawm lub hauv pliaj, tsis muaj zog, tshee ntawm tes, mob taub hau, loog ntawm daim di ncauj. Tus neeg mob yog txhoj puab heev, tsis zoo tswj nws lub zog, muaj lub plawv dhia, ob lub zeem muag, tsis meej pem. Nyob rau theem tom ntej, muaj kev tsis nco qab, convulsions. Nyob rau theem prehospital, cov neeg mob no tuaj yeem muab dej qab zib lossis ib yam dab tsi noj, sov thiab thauj mus rau qhov chaw kho mob rau kev saib xyuas.

xov xwm lig

Cov teeb meem no tshwm sim hauv cov ntshav qab zib decompensated tom qab muaj mob ntau xyoo. Lawv qhov txaus ntshai nyob rau hauv qhov tseeb tias lawv maj mam thiab imperceptibly ua phem rau tus neeg mob tus mob. Tsis tas li ntawd, feem ntau txawm tias muaj peev xwm kho mob ntshav qab zib tsis tuaj yeem lav tias sai lossis tom qab lawv yuav tsis tshwm sim.

  1. YRetinopathycharacterized los ntawm kev puas tsuaj rau retinal. Hmoov tsis zoo, txawm tias tag nrho cov kev ua tiav ntawm cov tshuaj niaj hnub, qhov tshwm sim ntawm retinopathy nyob rau hauv cov neeg mob uas muaj hom 2 mob ntshav qab zib mellitus, uas kav ntev dua 20 xyoo, nyhav rau 100%. Qhov no pathology ua rau hemorrhages nyob rau hauv lub fundus thiab nws thiaj li ua rau kom tiav tsis pom kev.
  2. Angiopathies - ua txhaum ntawm vascular permeability, uas ua rau kev loj hlob ntawm thrombosis thiab atherosclerosis. Muaj peev xwm txhim kho nyob rau lub sijhawm luv luv (kwv yees li ib xyoos), tsis zoo li lwm yam teeb meem lig uas feem ntau ua rau cov keeb kwm kho mob hnyav dua.
  3. Hom 2 mob ntshav qab zib, ntshav qab zib polyneuropathy, uas kuj yog ib qho teeb meem tshwm sim, sau npe hauv 90% ntawm cov neeg mob. Clinically, qhov no yog qhia los ntawm kev puas tsuaj rau cov hlab ntsha uas tswj ob leeg leeg thiab cov kabmob hauv nruab nrog cev. Polyneuropathy tuaj yeem tshwm sim los ntawm kev kub siab, nrawm nrawm (tua) mob, kub hnyiab. Tsis tas li ntawd, kev tshwm sim ntawm neuropathy tuaj yeem ua rau qaug zog, txo qis rhiab heev, uas ua rau muaj kev raug mob ntau heev. Ntawm cov kab mob hauv nruab nrog cev, tej zaum yuav muaj raws plab, urinary incontinence, mob taub hau, kiv taub hau, cuam tshuam kev ua haujlwm ntawm lub plawv.
  4. Mob ntshav qab zib ko taw - yog tus cwj pwm los ntawm cov tsos ntawm ob txhais ceg thiab, tshwj xeeb, ntawm ko taw ntawm tus neeg mob ntshav qab zib trophic ulcers, abscesses, thaj chaw necrotic. Tias yog vim li cas cov neeg mob uas raug kev txom nyem los ntawm cov kab mob no tau pom zoo kom them nyiaj tshwj xeeb rau kev xaiv khau.

Huab cua

Kev kho mob keeb kwm hom 2 mob ntshav qab zib mellitus subcompensation
Kev kho mob keeb kwm hom 2 mob ntshav qab zib mellitus subcompensation

Sad as it may seem, butQhov tseeb yeej swb ntshav qab zib mellitus cov tshuaj niaj hnub no tsis muaj peev xwm. Txawm li cas los xij, nws tuaj yeem kho raws sijhawm raws li qhov sib txawv uas tshwm sim hauv tib neeg lub cev los ntawm kev nce siab ntawm cov piam thaj, uas yog, hom 2 mob ntshav qab zib mellitus. Keeb kwm ntawm tus kab mob, qhov kev tshwm sim ntawm tus kab mob no nyob rau hauv ntau txoj kev, yog tias tsis nyob rau hauv txhua yam, yuav nyob ntawm seb tus neeg mob tus cwj pwm ntawm nws. Nrog ceev faj saib xyuas ntawm nws cov theem ntawm cov piam thaj thiab lwm yam ntsuas, kev kho tshuaj tsim nyog, kev noj haus thiab cov lus pom zoo ntawm cov kws kho mob, cov teeb meem tuaj yeem zam tau ntau xyoo.

Pom zoo: