Coded as K98.1 hauv ICD 10, tshuaj tiv thaiv kab mob raws plab (AAD) yog ib qho mob plab uas tsis cuam tshuam nrog kev kis kab mob lossis lwm yam ua rau. Qhov mob no yog ua ntej los ntawm kev siv tshuaj tua kab mob. Kev zawv plab tau hais tias tshwm sim yog tias cov quav xoob tau pom peb zaug ob hnub ua ke lossis ntau dua. Qee zaum ABP tau kho qee lub sijhawm tom qab kawm tiav chav kho mob - mus txog yim lub lis piam.
saib dav dav
Kev nkag mus los ntawm cov cim K98.1 hauv ICD, cov tshuaj tiv thaiv kab mob sib kis hauv cov kab mob tsis tshua muaj peev xwm tiv thaiv keeb kwm yav dhau los ntawm kev kis tus kab mob, tab sis feem ntau piav qhia los ntawm kev cuam tshuam ncaj qha ntawm cov tshuaj nyob rau hauv txoj hnyuv motility lossis indirect cawv. Tsis tas li ntawd, cov tshuaj muaj kev cuam tshuam rau ntau qhov chaw ntawm lub plab zom mov, uas tuaj yeem ua rau cov quav tsis muaj kab mob. Ib qho piv txwv zoo yog macrolides, uas muaj cov nyhuv zoo li motilin. Kev kho mob nrog tshuaj nrog ceftriaxone tuaj yeem ua tauua rau sludge syndrome. Cov tsos mob ntawm tus kab mob pathological nyob rau hauv daim ntawv ntawm qhov teeb meem no ploj ntawm lawv tus kheej qee lub sij hawm tom qab tso tseg cov tshuaj. Tsis tas yuav muaj ib txoj haujlwm tshwj xeeb los kho tus neeg mob tus mob.
K98.1 - ICD 10 code rau tshuaj tua kab mob raws plab, uas yog, quav quav vim yog kev kho mob ntawm cov tshuaj tua kab mob. Raws li tuaj yeem pom los ntawm cov ntaub ntawv kho mob thiab kev txheeb xyuas kev kho mob, yuav luag 37% ntawm cov neeg mob uas raug yuam kom noj cov tshuaj hauv pab pawg no tau pom qhov tshwm sim ntawm AAD, uas tau sau tseg hauv lawv cov keeb kwm kho mob. Qhov kev qhia ntau zaus yog qhov kwv yees yam tsawg kawg nkaus rau cov neeg mob plab hnyuv, tab sis qee cov kws tshaj lij ntseeg tau tias qhov teeb meem muaj ntau dua. Kev kwv yees tsis yog tag nrho ntawm tus lej ntawm tus neeg mob yog cuam tshuam nrog kev ntsuas siab ntev ntawm kev tshwm sim - ob tus neeg mob thiab kws kho mob tsis pom qhov tshwm sim raws li kev mob tshwm sim. Qhov no yog qhov tshwj xeeb tshaj yog tias qhov ua txhaum ntawm cov quav tau pom nyob rau hauv daim ntawv me me lossis nruab nrab ntawm qhov hnyav.
Shapes and nuances
ICD code rau kab mob raws plab K98.1 suav nrog ntau hom kev kho mob ntawm cov kab mob pathological. Tsis ntev los no, cov txheej txheem kev faib tawm tau siv dav, suav nrog kev ntsuas ntawm qhov tshwm sim. Muaj AAD tsis muaj cov tsos mob ntawm colitis, AA colitis thiab pseudomembranous. Thaum kis tau qee yam ntawm Clostridium, AAD raug cais tawm tias tsis muaj cov tsos mob ntawm tus kab mob plab, thiab tseem muaj peb yam ntawm nws: fulminant, pseudomembranous, thiab ib daim ntawv tsis muaj pseudomembranes.
Txog 20% ntawm txhua kis yog vim Clostridiumhom nyuaj. Txoj cai K98.1 siv nyob rau hauv ICD rau cov tshuaj tiv thaiv kab mob raws plab kuj suav nrog lwm tus neeg mob, uas suav txog (kwv yees) txog 80% ntawm tag nrho cov neeg mob. Cov no yog cov xwm txheej uas ua txhaum ntawm cov quav nrog rau lwm hom clostridium, fungal microflora, cocci, salmonella, klebsiella. Qhov kawg, raws li tau tsim, feem ntau ua rau cov kab mob hemorrhagic pathological ntawm lub plab zom mov.
Kev kuaj mob thiab kev faib tawm
Nyob rau xyoo 2009, cov kws kho mob tshwj xeeb, cov kws kho mob microbiologist, cov tswv cuab ntawm European Union ntawm Cov Kws Kho Mob, tau tshaj tawm cov lus pom zoo kho mob uas cuam tshuam rau cov tshuaj tiv thaiv kab mob raws plab. Ib qho txiaj ntsig zoo ntawm kev ua haujlwm tshawb fawb tau mob siab rau feem ntau ntawm cov kab mob microflora - Clostridium difficile. Cov teeb meem ntawm kev kuaj mob thiab kev kho mob ntawm cov xwm txheej zoo li no tau txiav txim siab. Cov kws kho mob tshwj xeeb tau them nyiaj tshwj xeeb rau kev soj ntsuam ntawm qhov mob hnyav ntawm tus neeg mob, kev tsim kho ntawm cov kab mob. Ib xyoos tom qab, Asmeskas kws kho mob kis mob tau tshaj tawm cov lus pom zoo rau kev saib xyuas, kho cov neeg mob nrog AAD, uas daim ntawv no ntawm microflora dominates.
Qhov teeb meem ceev ceev ntawm kev mob raws plab tom qab siv tshuaj tua kab mob hauv cov neeg laus thiab menyuam yaus cuam tshuam nrog kev paub tsis txaus ntawm qhov teeb meem. Tshwj xeeb, rau hom kab mob ntawm lub neej uas tau hais los saum no, ib hom kab mob tshiab tau pom tsis ntev los no, uas yog tus cwj pwm los ntawm kev tsim cov tshuaj lom ntau ntxiv hauv kev sib piv nrog cov neeg paub ua ntej. Qhov sib txawv mus txog 23 zaug. Kev kis tus kab mob no ua rauhnyav AAD. Ntawm cov khoom tsim los ntawm microflora yog binary toxin. Cov kev ntsuas uas tau coj los txog tam sim no tseem tsis tau tso cai qhia meej tias cov tshuaj no cuam tshuam dab tsi rau tus neeg. Ib qho tshwj xeeb ntawm hom kev txheeb xyuas yog ua kom muaj zog tiv thaiv fluoroquinolones. Los ntawm qhov no, cov kws kho mob tau xaus lus tias kev siv fluoroquinolones tuaj yeem yog ib qho ntawm qhov ua rau AAD.
Nuances and manifestations
Tshuaj tua kab mob sib txuas nrog dysbacteriosis, AAD tuaj yeem txhim kho ntau hom. Qee cov neeg mob muaj raws plab me me uas daws tau sai. Lwm tus raug kuaj pom tias muaj mob plab hnyav, cuam tshuam nrog kev phom sij ntawm kev tuag. Hauv feem pua ntawm cov neeg mob feem ntau, qhov tshwm sim yog qhia los ntawm kev tsis muaj zog ntawm cov zais zis, tsis muaj zog tshwm sim ntawm colitis. Tsis muaj cov tsos mob dav dav. Lub rooj zaum tshwm sim txog plaub zaug hauv ib hnub, nrog rau kev mob me ntsis, zoo li kev cog lus, hauv plab. Qhov kub tseem nyob li qub. Ntawm palpation, hypersensitivity tuaj yeem txiav txim siab, tab sis tsis tas li. Kev tsim roj kuj tseem muaj zog dua li qub, tab sis qhov sib txawv ntawm lub xeev noj qab haus huv tsis loj heev.
Tshuaj tua kab mob raws plab hauv cov menyuam yaus thiab cov neeg laus tsis pom nws tus kheej ua cov cim ntawm kev mob hauv lub cev. Cov tsos mob feem ntau yog txo qis los ntawm kev noj tshuaj tshwj xeeb, tshem tawm cov tshuaj tiv thaiv kab mob. Txhawm rau txhim kho tus mob sai sai, nws raug nquahu kom siv cov tshuaj probiotics, tshuaj tiv thaiv kab mob raws plab. Cov kws kho mob tau tsim muaj tseeb: qhov mob no cuam tshuam nrog kev tsis sib haum xeeb hauv cov hnyuv microflora,kev ua haujlwm tsis zoo ntawm cov kab mob muaj txiaj ntsig. Tsis muaj kev loj hlob ntawm pathological microscopic life forms.
Instances: qee zaum nyuaj
Kev kho mob raws plab tom qab tshuaj tua kab mob sib txawv yog tias qhov kev kho mob tshwm sim ntawm Clostridium difficile-associated AAD nyob rau hauv cov ntaub ntawv ntawm colitis muaj kev txhawj xeeb. Xws li ib tug mob yuav xav tias muaj zog, unpleasantly tsw tsw, nyob rau hauv uas mucous inclusions yog pom. Lub rooj zaum yog profuse. Lub plab zom mov yog nrog los ntawm tenesmus. Tus neeg mob muaj kev txhawj xeeb txog qhov mob, zoo li kev cog lus, hauv plab. Ntawm palpation, qhov chaw no yog mos, qee qhov chaw teb nrog nce rhiab heev (colon). Kev mloog tso cai rau koj los txiav txim: suab nrov hauv cov hnyuv ntau dua li qub.
Yog hais tias raws plab tom qab noj tshuaj tua kab mob cuam tshuam nrog cov txheej txheem ntawm lub neej, tus neeg mob kub taub hau (qhov mob hnyav heev). Muaj lub cev qhuav dej ntawm lub cev, tus neeg mob xav tias muaj mob, ntuav. Kev kuaj ntshav qhia tau hais tias qhov tsis tseem ceeb leukocytosis txawm tias tsis muaj qhov tshwm sim ntawm raws plab. Colitis feem ntau yog nyob rau sab xis ntawm txoj hnyuv, qhia nws tus kheej nrog foci ntawm qhov mob, nce cov ntsiab lus ntawm leukocytes hauv cov ntshav, thiab kub taub hau. Mob plab me me los yog tsis tuaj.
Variants thiab case
Qee zaum raws plab tom qab noj tshuaj tua kab mob hnyav heev. Toxic megacolon nrog rau cov quav tsis tshua muaj. Hauv kev kho mob, cov xwm txheej ntawm qhov kev ntsuas tsis txaus ntawm qhov kev nce qib hauv tus neeg mob tus mob tau paub - qee zaum cov kws kho mob (thiab cov neeg mob lawv tus kheej) coj cov tsos mob los ua ib qho kev txhim kho. Nyob rau tib lub sijhawm, hauvgases khaws cia nyob rau hauv txoj hnyuv ib ntsuj av, lub peritoneal cheeb tsam yog khaus, tus neeg yog kub taub hau, kev tshawb fawb qhia ib tug distension ntawm txoj hnyuv. Kev kawm ntxaws ntxaws ntawm tus neeg mob tus mob yuav pab kom pom cov effusion hauv peritoneum, lub plab me me. Nyob rau hauv lub circulatory system, ib tug nce qib ntawm leukocytes yog tsim, thiab cov concentration ntawm albumins, ntawm qhov tsis tooj, yog qis dua qub. Tsis tas li ntawd, hypovolemia raug kuaj pom. Cov kev tshwm sim no yog ib daim duab kho mob.
Yog tias mob raws plab nrog tshuaj tua kab mob tshwm sim hauv daim ntawv no, tus neeg mob yuav tsum raug xa mus rau x-ray. Hauv kev lom megacolon, txoj hnyuv loj nthuav dav. Kev tshawb fawb pab txheeb xyuas cov kab mob hauv plab hnyuv. Tom qab CT, nws muaj peev xwm tsim kom muaj cov phab ntsa hauv plab hnyuv, nqaim ntawm lumen, compaction ntawm fatty structures nyob ib ncig ntawm txoj hnyuv, nrog rau ascites. Tus mob no mob hnyav heev, yog li kev ua kom tsis zoo rau theem ntawm kev zawv plab tom qab siv tshuaj tua kab mob yuav tsum tau zam. Yuav ua li cas yog tias qhov kev vam meej tau mus txog theem no, cov kws kho mob tsim nyog paub: tus neeg mob tau pom tias muaj kev ua haujlwm ceev. Txawm li cas los xij, raws li tuaj yeem pom los ntawm kev kho mob, feem pua ntawm cov neeg mob tau ntsib nrog cov teeb meem loj heev, qhov tshwm sim tsis zoo ntawm kev cuam tshuam. Nce kev tuag.
Teeb meem: cov xwm txheej raug
Yog tias ua ntej cov tshuaj tiv thaiv kab mob raws plab tau piav qhia los ntawm cov ntaub ntawv muaj kev nyab xeeb ntawm pathological microflora, tsis ntev los no cov kab mob txaus ntshai tshaj plaws ntawm clostridia, BI / NAPI, tau dhau los ntau zaus. Ntau zausFeem ntau qhov tshwm sim ntawm hom AAD no tau pom nyob hauv tsev kho mob, qhov twg cov neeg mob raug yuam kom mus kawm cov kev kho mob ntev ntev. Xws li AADs hnyav dua li lwm hom thiab hom kab mob.
Feem ntau, cov tsos mob pib tshwm sim hnub thib tsib tom qab pib qhov kev pabcuam tshuaj tua kab mob, qee zaum nws yuav siv sijhawm ob zaug ntev ua ntej qhov tshwm sim thawj zaug. Cov kab mob sib cais ntawm AAD cov tsos mob tau paub lawm nyob rau hnub thib ob ntawm kev noj tshuaj, tab sis kuj tseem muaj qhov txawv txav, thaum thawj qhov tshwm sim tshwm sim nyob rau lub lim tiam thib kaum tom qab kawg ntawm kev kho tshuaj tua kab mob.
BI/NAPI: Mild AMA
Tshuaj tiv thaiv kab mob raws plab ntawm hom no yog tshwm sim los ntawm kev txo qis hauv kev ua haujlwm ntawm cov hnyuv me thiab kev sib kis ntawm lub cev. Lub plab zom mov ntawm cov zaub mov tsis muaj zog, fermentation, lwj nrog kev koom tes ntawm cov kab mob ua kom nquag plias. Cov kua qaub ntawm cov ntsiab lus ntawm txoj hnyuv poob, vim qhov kev ua ntawm lipase tsis meej pem. Tus neeg mob muaj steatorrhea, xab npum tebchaw thiab fatty qauv yog tsim nyob rau hauv txoj hnyuv. Cov tshuaj muaj roj-soluble vitamin absorbed ntau zuj zus, uas provokes endogenous polyhypovitaminosis.
Vim tias adsorption thiab digestive txheej txheem nyob rau hauv cov hnyuv me raug cuam tshuam, cov tshuaj tiv thaiv kab mob raws plab ua rau muaj roj active tsim thiab imbalance ntawm txoj hnyuv motility, ua rau ib tug tsis tu ncua dyspepsia syndromic complex. Kev ua haujlwm ntau dhau ntawm cov organic acids vim yog kev cuam tshuam los ntawm microflora ua rau nce osmolarity ntawm obsessed gastrointestinal ib ntsuj av. Qhov tshwm sim ntawm qhov tshwm sim yog tsam plab, flatulence, raws plab, bursting mob, uas los ntawm kev tawm tsam. Dysbiosis provokes qib siab permeability ntawm txoj hnyuv barrier, uas pib ib qho kev tsis haum tshuaj ntawm lub cev. Kev loj hlob ntau dhau ntawm microflora hauv cov hnyuv tuaj yeem ua rau tsis muaj zog ntawm kev ua haujlwm ntawm lwm qhov ntawm lub plab zom mov, vim li ntawd - nce siab, duodenostasis, IBS, pseudo-obstruction. Nws yog ib qho ua tau los pib cov txheej txheem inflammatory vim kev sib kis mus ntev, deconjugation. Enteritis lossis duodenitis raug kaw rau hauv tus neeg mob daim npav.
Kev txiav txim siab txuas ntxiv
Tshuaj tua kab mob raws plab txuam nrog Clostridium difficile tsis tas yuav kho yog tias nws mob me. Nws tsis yog qhov tsim nyog los kho tus neeg mob tus mob yog tias qhov mob me me, thiab kev tshem tawm tau kho txog plaub zaug hauv ib hnub, thaum tsis muaj cov tsos mob tshwm sim, kev tshawb fawb hauv chav kuaj pom tias tsis muaj kev hloov pauv tseem ceeb. Yog tias tus mob no tshwm sim hauv tsev, nws yog txwv tsis pub siv tshuaj tua kab mob kom tshem tau AAD.
raws li txoj cai, raws plab ua tiav cov kev daws teeb meem ntawm nws tus kheej thaum tus neeg mob ua tiav txoj kev kho no uas ua rau nws. Qee zaum, tus kws kho mob yuav pom zoo kom noj cov tshuaj probiotics. Yog tsis muaj kev pom zoo los ntawm kev kho mob, yuav tsum tsis txhob siv tshuaj, thiaj li tsis ua rau muaj kev puas tsuaj rau tus mob.
BI/NAPI: Severe AAD
Qee zaum, AAD tau ua raws li qhov xwm txheej tsis zoo, colitis tshwm sim. Muaj ob lub ntsiabCov ntaub ntawv: nrog pseudomembrane thiab tsis muaj lawv. Yog tsis muaj pseudomembranes, cov txheej txheem feem ntau yog systemic. AAD manifests nws tus kheej raws li lub xeev kub taub hau, lom lom ntawm lub cev, thiab mob plab. Tus neeg mob xeev siab thiab ntuav. Cov quav nquag, dej. Npua txog nees nkaum zaug hauv ib hnub yog ua tau. Muaj lub cev qhuav dej.
Pseudomembranous colitis pib tshwm sim nrog cov tsos mob zoo sib xws. Colonoscopy qhia pseudomembrane. Thaum lub sij hawm coproscopy, erythrocytes thiab leukocytes tuaj yeem kuaj pom. Kev kuaj ntshav occult hauv feem pua ntawm cov neeg mob feem ntau muab qhov txiaj ntsig zoo. Qee zaum muaj hematochezia.
Qhov kev hloov loj tshaj plaws ntawm cov kab mob pathological yog fulminant colitis. Nws tshwm sim hauv kwv yees li 3% ntawm cov neeg mob. Cov mob tuaj yeem ua rau plab hnyuv, megacolon tiv thaiv keeb kwm yav dhau los ntawm kev lom, plab hnyuv perforation, o hauv plab kab noj hniav, ntshav lom. Fulminant colitis tuaj yeem xav tias yog tus neeg mob raug mob los ntawm qhov kaj, qhov mob zoo hauv plab thiab tsam plab. Colitis yog nrog los ntawm lub cev qhuav dej, kub taub hau, hypotension, kev nyuaj siab ntawm kev nco qab los yog ntxhov siab. Toxin A, tsim los ntawm pathological microflora, ncaj qha tshuaj lom rau hauv nruab nrab paj hlwb, uas tuaj yeem ua rau mob encephalopathy.
Case development: mloog rau nuances
Thaum AAD tuaj yeem pom qhov tshwm sim, qhia kev khaus ntawm cov ntaub so ntswg ntawm peritoneum. Tej zaum cov leeg nro hauv qee qhov chaw. Cov xwm txheej zoo li no yog lub hauv paus rau kev tawm tswv yim plab hnyuv perforation. Hauv kev tshawb fawb hauv chav kuaj, nws muaj peev xwm tsim kom muaj kev nce ntxivQhov concentration ntawm leukocytes hauv cov ntshav, azotemia.
Kev mob plab hnyuv, tshuaj lom megacolon, uas tus mob tuaj yeem ua tiav, ua rau cov quav tsis tshua muaj. Qee zaum colitis yog tshwm sim los ntawm lub plab syndrome nyob rau hauv daim ntawv mob, tab sis tsis nrog raws plab. Qhov no kuj tseem ua tau nrog megacolon tiv thaiv keeb kwm ntawm kev lom ntawm lub cev.
Tsis yog tus qauv
Atypical AAD tuaj yeem txhim kho. Nrog rau daim ntawv no ntawm tus kab mob, tus neeg mob raug kev txom nyem los ntawm colitis, kev ncaj ncees thiab kev noj qab haus huv ntawm cov hnyuv me yog cuam tshuam. Muaj qhov poob ntawm cov qauv protein, enteropathy. Kev soj ntsuam tus neeg mob tus mob tso cai rau koj txheeb xyuas cov tsos mob ntawm plab hnyuv.
Kev piav qhia
Rau cov tsos mob ntawm AAD, colitis, suav nrog cov neeg xav tias muaj feem cuam tshuam nrog cov kab mob tshiab tshaj plaws thiab txaus ntshai tshaj plaws ntawm Clostridium, yuav tsum muaj keeb kwm kho mob. Yog tias ib tus neeg tau siv tshuaj tua kab mob hauv ob lub hlis dhau los, nws yuav tsum xav tias qhov yuav tshwm sim ntawm AAD yog qhov siab dua qhov nruab nrab. Hauv kev kuaj mob sib txawv, nws yog ib qho tsim nyog los txiav txim siab qhov nuances ntawm chav kawm ntawm rooj plaub. Nws yog ib qho tseem ceeb uas yuav tsum tau kuaj cov quav, ntshav, zis rau kev kuaj xyuas, thiab ua qhov kev kuaj mob hauv chav kuaj. Nws yog ib qho tsim nyog los xyuas qhov tseeb ntawm tus kab mob clostridial. AAD yog qhia los ntawm qhov tsis muaj albumin, azotemia, cov ntsiab lus ntawm leukocytes - 15-16 txhiab ib hli cu.
Yog xav tias mob plab, ua ntej ntawm tag nrho cov, nws yog ib qho tsim nyog yuav tsum tau x-ray, soj ntsuam cov kab mob hauv plab. Kev kuaj mob tau lees paub los ntawm kev tshawb pom ntawm perforation, megacolon, pneumatosis, ileus. CT tuaj yeem pom qhov tuab tuabplab hnyuv phab ntsa nyob rau hauv nyias qhov chaw, ascites. Tsawg feem ntau obliteration, plab hnyuv perforation raug kuaj.
Txoj kev kuaj mob kom raug thiab ceev tshaj plaws yog kev tshuaj xyuas cov quav kom pom cov kab mob. Rau qhov no, kev tshawb fawb tau ua los txheeb xyuas cov ntsiab lus ntawm cov tshuaj lom A. Immunological enzymes tau siv. Qhov tseeb thiab rhiab heev ntawm cov tshuab ntsuas niaj hnub yog kwv yees li ntawm 75-85%. Cov txheej txheem tau tsim los rau tib lub sijhawm tshawb xyuas cov co toxins A, B. Txoj hauv kev no suav tias yog qhov tseeb dua.
Endoscope kom paub meej qhov xwm txheej
Txoj kev tshawb no yog ceev faj tshaj plaws yog tias muaj laj thawj ntseeg tias cov tshuaj tiv thaiv kab mob raws plab uas tau nce mus rau colitis xav tau kev kho mob. Nrog xws li kev nce qib, cov txheej txheem suav hais tias yog qhov txaus ntshai, vim tias nws ua rau muaj kev pheej hmoo ntawm plab hnyuv perforation. Rau qhov ntau dua, qhov no yog tus yam ntxwv ntawm cov mob hnyav.
Yog tias pseudomembranous colitis tau tsim, kev kuaj kab mob plab tau lees paub tias yog txoj hauv kev txhim khu kev qha tshaj plaws kom paub meej qhov kev kuaj mob. Muab qhov kev pheej hmoo siab cuam tshuam nrog cov xwm txheej zoo li no, kev tshuaj xyuas tsuas yog ua tiav hauv rooj plaub thaum nws tsim nyog los txiav txim siab qhov kev kuaj mob sai sai thiab nrog qhov tseeb tshaj plaws, nrog rau cov ntaub ntawv ntawm ileus. Colonoscopy yog qhov tsim nyog los sib txawv ntawm tus mob thiab tsis suav nrog lwm cov kab mob pathological ntawm txoj hnyuv uas cuam tshuam rau tus neeg mob lub neej.
Yuav ua li cas?
Cov kws kho mob zoo tshaj plaws paub kho mob raws plab tom qab siv tshuaj tua kab mob. Feem pua ntawm cov tib neeg feem ntau tau ntsib nrog daim ntawv me me ntawm AAD, yog li ntawd, qhov tshwj xeebtsis tas yuav kho. Cov tsos mob ploj mus ntawm lawv tus kheej thaum kawm tiav cov tshuaj tiv thaiv kab mob. Qee lub sij hawm kho cov tsos mob yog muab los tiv thaiv lub cev qhuav dej, kho qhov sib npaug ntawm electrolytes hauv lub cev. Yog tias cov tsos mob qhia tias mob plab, yuav tau muab tshuaj tua kab mob.
Hauv kev tsim cov lus pom zoo txog kev kho mob raws plab tom qab siv tshuaj tua kab mob thaum nqa Clostridium difficile yam tsis muaj cov tsos mob tshwm sim, cov kws kho mob hauv Asmeskas tau txiav txim siab tias nws tsis tas yuav muab tshuaj rau tus neeg mob los kho tus mob tshwj xeeb. Feem ntau, lawv ua tiav cov kab mob tua kab mob thiab tsis siv txoj hauv kev los tiv thaiv kev ua haujlwm secretory, plab hnyuv motility - lawv muaj peev xwm provoke active reproduction ntawm pathological microflora.
Kev kho mob tseem ceeb yog kev siv cov tshuaj probiotics, uas yog, cov kab mob muaj sia uas rov ua kom qhov sib npaug ntawm microflora hauv plab hnyuv. Cov no yog cov kab mob ntau yam: lacto-, bifido-, sticks, cocci, fungal kab lis kev cai. Ntau tus kws tshawb fawb tau ntseeg tias probiotics tuaj yeem siv los tiv thaiv AAD. Cov lus nug no tau qhib tam sim no, ntau cov kev tshawb fawb tau ua tiav los lees paub qhov kev xav lossis tsis lees paub nws.
